Mitochondria are a source of ROS and free radicals in mammalianoocytes, even under physiological conditions (Takahashi et al., 2013).Despite the fact that ROS are essential participants in a wide range of cellular processes (Bergamini et al., 2004), the inability of the antioxidantdefense to quench an increased production of ROS creates a biochemicalimbalance known as oxidative stress. Such oxidative stress is capable ofdisrupting many cellular components and activities, possibly resulting indevelopmental blockage (Velez-Pardo et al., 2007; Agarwal et al.,2012) and cell death (Karlsson et al., 2010).
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